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Cardio-selective Beta-adrenergic Therapy Report

Beta adrenergic therapy with propranolol is the therapy of choice in the treatment of hypertrophic obstructive cardiomyopathy. However, it is contraindicated in the presence of severe asthma. Recendy a cardio-selective β-1 adrenergic antagonist, pindolol, or 4-(2-hydroxy-3-isopropylaminopropoxy) indole has been developed with a potency equal to or greater than that of propranolol. Specific β-2 adrenergic agonists also are available for use in patients with asthma. Their effects on the cardiovascular system are minimal. We recently encountered

Beta-Adrenergic Blockade with Pindolol (LB-46) in Mild to Moderate Asthma Observations

Acceptable beta-adrenergic antagonists for use in patients with asthma should ideally minimize antagonism of bronchial (beta2) receptors while providing useful cardiac (beta2) blockade. Propranolol, a nonselective beta-adrenergic blocking drug, aggravates bronchospasm in asthmatics . Metoprolol, a selective betai blocker approved for use in the United States, may have decreased effect on the airways. However, its selectivity is thought to be dose-related and perhaps limited to the lower dose ranges. Pindolol (LB-46), a potent beta-adrenergic blocker, does not have

Beta-Adrenergic Blockade with Pindolol (LB-46) in Mild to Moderate Asthma

Characteristics of Study Group Subject characteristics are shown in Table 1. All had mild to moderate asthma as shown by comparison of FEV1/FVC and the FEV1/FEV1 predicted. There were 13 male and 11 female subjects. As noted, all had at least 10 percent improvement in FEVi following inhalation of isoproterenol. Presence of Exercise-Induced Bronchospasm We compared postexercise with preexercise pulmonary function for the placebo trial to demonstrate the presence of exercise-induced bronchospasm . Comparisons of the

Labetalol and Hydrochlorothiazide for Ventilatory Function of Hypertensive Patients Outcomes and Deliberations

Read the article with this research initial data described. Patient Characteristics Of the 41 patients who entered the treatment phase of the study, 21 received therapy with labetalol and 20 received hydrochlorothiazide treatment (Table 1). The ratio of men to women was similar in both groups. Mean age was 54 years in the labetalol group and 59 years in the hydrochlorothiazide group. The preponderance of middle-aged males reflects the fact that two of the participating hospitals were Veterans

Labetalol and Hydrochlorothiazide for Ventilatory Function of Hypertensive Patients Research

Beta adrenergic antagonists are widely used antihypertensive agents with therapeutic effectiveness similar to that of methyldopa and thiazide diuretics. Their advantages over other antihypertensive medications include a lack of postural hypotension, effectiveness in the supine position, and their potential cardioprotective ability. An important consideration with currently available beta-adrenergic antagonist drugs is their ability to block the betas adrenergic receptors in the lungs, resulting in worsened ventilatory function in patients with obstructive lung disease. Two currently

PY108-068, a New Calcium Antagonist, with Nifedipine in Exercise-Induced Asthma Deliberations

Part 1: Research about PY108-068, a New Calcium Antagonist, with Nifedipine in Exercise-Induced Asthma Part 2: PY108-068, a New Calcium Antagonist, with Nifedipine in Exercise-Induced Asthma Outcomes Bronchodilation and prevention of exercise-induced asthma are separate and useful goals of therapy for asthma. We studied a class of drugs that may have potential for meeting either or both of these goals. We found some resting bronchodilation associated with both 150 mg of PY 108-068 and nifedipine, although only the latter was significant. Some studies on calcium antagonists in asthma have not shown resting bronchodilation; however, measurements were only made

PY108-068, a New Calcium Antagonist, with Nifedipine in Exercise-Induced Asthma Outcomes

Article shows research outcomes based on data which is given in this article . Twelve of 16 subjects completed the study. Four subjects dropped out after the first day (two due to exacerbations of asthma thought unrelated to the study, one due to lack of cooperation, and one due to an unrelated illness). Resting bronchodilation, expressed as mean percentage of change in FEW from baseline, is shown in Figure 1. The peak bronchodilator effect of nifedipine occurred at

Research about PY108-068, a New Calcium Antagonist, with Nifedipine in Exercise-Induced Asthma

Many of the pathophysiologic events underlying asthma, including contraction of bronchial smooth muscle and degranulation of mast cells, are calcium-dependent. Because of this, studies have been done to determine the effect of calcium antagonists on pulmonary function in asthmatic subjects. These studies have measured the effect of these drugs on flow rates at rest and after various challenges including antigen histamine, hyperventilation, and exercise. Exercise has frequently been used because it is safe, natural, and

Bronchial Coarctation Considerations

Case Report of Bronchial Coarctation is published here . Chevalier Jackson, describing lesions he observed through a bronchoscope, noted that a few patients with asthma had “ organic stenosis of the bronchi .” He felt that in children some of the lesions were congenital, whereas in adults, they were “fibrotic” and could be treated by dilation. It is unlikely that our patient has asthma since she did not improve significantly after treatment with 40 mg of prednisone a day for eight

Bronchial Coarctation Report

Severe narrowing of large bronchi may occur in chronic bronchitis, asthma ( https://onlineasthmainhalers.com/category/asthma ), tuberculosis, sarcoidosis, and amyloidosis. In chronic bronchitis, these changes are said to occur “distal to the third or fourth generation of bronchi.” We have observed a patient with chronic bronchitis who had irreversible bronchial narrowing in her proximal third and fourth order bronchi. Case Report A 38-year-old white woman developed constant shortness of breath without episodic remissions in early 1975. She had